高原

发布时间：2025-09-14浏览次数：2509

  • 1、Melatonin ameliorates neurological deficits through MT2/IL-33/ferritin H signaling-mediated inhibition of neuroinflammation and ferroptosis after traumatic brain injury,Free Radic Biol Med,SCI,2023.04,高原,苏州大学,199: 97-112,1
  • 2、Extended characterization of IL-33/ST2 as a predictor for wound age determination in skin wound tissue samples of humans and mice.,Int J Legal Med,SCI,2023.07,高原,苏州大学,137(4):1287-1299.,1
  • 3、TrkB agonist N-acetyl serotonin promotes functional recovery after traumatic brain injury by suppressing ferroptosis via the PI3K/Akt/Nrf2/Ferritin H pathway,Free Radic Biol Med,SCI,2022.01,程颖,苏州大学,高原,194: 184-198,2
  • 4、Autophagy inhibition facilitates wound closure partially dependent on the YAP/IL-33 signaling in a mouse model of skin wound healing.,FASEB J,SCI,2021.10,高原,苏州大学,35 (10):e21920,1
  • 5、Emerging Effects of IL-33 on COVID-19.,Int J Mol Sci,SCI,2022.11,高原,苏州大学,23 (21),1
  • 6、Emerging functions and therapeutic targets of IL-38 in central nervous system diseases,CNS Neuroscience & Therapeutics,SCI,2023.12,高原,苏州大学,1
  • 7、IL-33 Exerts Neuroprotective Effect in Mice Intracerebral Hemorrhage Model Through Suppressing Inflammation/Apoptotic/Autophagic Pathway.,Mol Neurobiol,SCI,2017.07,高原,苏州大学,54(5):3879-3892,1
  • 8、IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure.,Frontiers in molecular neuroscience,SCI,2017.12,高原,苏州大学,10:423,1
  • 9、IL-33/ST2L Signaling Provides Neuroprotection Through Inhibiting Autophagy, Endoplasmic Reticulum Stress, and Apoptosis in a Mouse Model of Traumatic Brain Injury.,Front Cell Neurosci,SCI,2018.04,高原,苏州大学,12: 95
  • 10、IL-33 Alleviated Brain Damage via Anti-apoptosis, Endoplasmic Reticulum Stress, and Inflammation After Epilepsy. ,Front Neurosci,SCI,2020.08,高原,苏州大学,14:898,1
  • 11、The Effects of YAP and Its Related Mechanisms in Central Nervous System Diseases.,Front Neurosci,SCI,2020.06,高原,苏州大学,14:595,倒1